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Pathology

Impaired production of β -chains leading to excess α-chains.

Aetiology

Autosomal recessive inherited genetic defect.
Most common inherited haematological disorder

Signs

Skeletal hypertrophy due to extramedullary haemopoiesis (frontal skull bossing, 
‘hair-on-end’ skull), hydramnios in pregnancy, severe anaemia, splenomegaly

Symptoms

β Thalassaemia trait (Heterozygotes): None or mild anaemia
β Thalassaemia major (Homozygotes): Failure to thrive, stunted growth, 

Investigations

Bloods: FBC, blood film, haemoglobin electrophoresis – Target Cells
Other: Genetic studies

Treatment

Medical: Folic acid, blood transfusion, desferrioxamine, bone marrow transplant
Surgical: Splenectomy

Prognosis

Variable depending on genes inherited and available supportive treatment
Haemosiderosis from repeated transfusions, recurrent infections, early death.


Key Facts

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Key References

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